Definition and research guide
This page combines a MeSH definition, publication statistics and selected research abstracts. It is a starting point for reading the literature, not a medical textbook or a systematic evidence review.
Content available: 4 complete abstracts (746 words), 10 references. Full article text is not stored or reproduced here; follow the PMID, DOI or PMC links to read the original sources and their access terms.
Definition
Protection conferred on a host by inoculation with one strain or component of a microorganism that prevents infection when later challenged with a similar strain. Most commonly the microorganism is a virus.
Definition: NLM Medical Subject Headings (MeSH) scope note (Public domain (U.S. National Library of Medicine); attribution requested). View the MeSH record.
Also known as: Protection, Cross.
Where it fits in the MeSH classification
- Broader topics
- Immunity
- See also
- Immunity, Heterologous
Research landscape
PubMed indexes 1,418 records tagged with the MeSH heading “Cross Protection” (counts retrieved 2026-10-29). 425 of them (30.0%) were published from 2020 onward, and the busiest period so far is 2010-2019 with 968 records.
Reviews account for 145 records (10.2%) and meta-analyses for 12. Randomized controlled trials number 25 (1.8%) as a share of all records on the topic. Randomized trials make up only a small share of this literature, so much of what is known comes from laboratory, observational or descriptive work.
Show the data
| Period | Records |
|---|---|
| through 1999 | 1 |
| 2000-2009 | 24 |
| 2010-2019 | 968 |
| 2020 onward | 425 |
Publication-type chart data
| Type | Records | Share |
|---|---|---|
| Reviews | 145 | 10.2% |
| Meta-analyses | 12 | 0.8% |
| Randomized controlled trials | 25 | 1.8% |
Method: PubMed query "Cross Protection"[MeSH Terms] with publication-type and date filters, retrieved 2026-10-29.
Reading the evidence in immunology
Immune responses are studied in model organisms, in cells from donors and in patients, and the same pathway can behave differently across these settings. Mouse immune systems differ from human ones in important ways, and blood measurements may not reflect what happens in tissues. Clinical conclusions about immune-mediated disease or immunotherapy come mainly from controlled trials and long-term cohorts. Check the species and tissue studied, whether markers were linked to outcomes patients notice, and the follow-up time.
Evidence from open-access studies
These open-access studies come from PubMed’s best-match results for this heading (papers whose main topic it is), ranked here by citation count, with one recent addition. Each abstract is reproduced in full under its recorded open licence so that you can read the authors’ own words. They illustrate the literature; they are not a systematic summary of it. Citation counts measure attention, not reliability or clinical benefit.
| Study / authors | Year | Source label / design | Europe PMC citations | Licence / source |
|---|---|---|---|---|
| Ancestral SARS-CoV-2-specific T cells cross-recognize the Omicron variant | 2022 | Research article Design not recorded in structured metadata | 401 | cc by PMID 35042228 · DOI · PMC8938268 |
| Cross-reactive serum and memory B-cell responses to spike protein in SARS-CoV-2 and endemic coronavirus infection | 2021 | Research article Design not recorded in structured metadata | 292 | cc by PMID 34011939 · DOI · PMC8134462 |
| BCG-Induced Cross-Protection and Development of Trained Immunity: Implication for Vaccine Design | 2019 | Review article Design not recorded in structured metadata | 265 | cc by PMID 31849980 · DOI · PMC6896902 |
| Cross-Protection in PRRSV: Mechanisms, Limitations, and Implications for Vaccine Design | 2026 | Review article Design not recorded in structured metadata | 7 | cc by PMID 42075672 · DOI · PMC13119244 |
Ancestral SARS-CoV-2-specific T cells cross-recognize the Omicron variant [1]
Abstract reading summary — source excerpts
- Opening excerpt
- The emergence of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) Omicron (B.
- Closing excerpt
- 529.
- Interpretation limits
- Verbatim excerpts from the authors’ abstract; the closing excerpt may describe a finding, limitation or recommendation. Design, population, effect size and uncertainty have not been independently extracted or appraised. Read the complete abstract and linked paper before interpreting the findings.
Read complete abstract (197 words)
The emergence of the severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) Omicron (B.1.1.529) variant of concern (VOC) has destabilized global efforts to control the impact of coronavirus disease 2019 (COVID-19). Recent data have suggested that B.1.1.529 can readily infect people with naturally acquired or vaccine-induced immunity, facilitated in some cases by viral escape from antibodies that neutralize ancestral SARS-CoV-2. However, severe disease appears to be relatively uncommon in such individuals, highlighting a potential role for other components of the adaptive immune system. We report here that SARS-CoV-2 spike-specific CD4+ and CD8+ T cells induced by prior infection or BNT162b2 vaccination provide extensive immune coverage against B.1.1.529. The median relative frequencies of SARS-CoV-2 spike-specific CD4+ T cells that cross-recognized B.1.1.529 in previously infected or BNT162b2-vaccinated individuals were 84% and 91%, respectively, and the corresponding median relative frequencies for SARS-CoV-2 spike-specific CD8+ T cells were 70% and 92%, respectively. Pairwise comparisons across groups further revealed that SARS-CoV-2 spike-reactive CD4+ and CD8+ T cells were functionally and phenotypically similar in response to the ancestral strain or B.1.1.529. Collectively, our data indicate that established SARS-CoV-2 spike-specific CD4+ and CD8+ T cell responses, especially after BNT162b2 vaccination, remain largely intact against B.1.1.529.
Abstract reproduced from the open-access article under the recorded cc by licence; © the authors. PMID 35042228 · DOI · PMC8938268
Cross-reactive serum and memory B-cell responses to spike protein in SARS-CoV-2 and endemic coronavirus infection [2]
Abstract reading summary — source excerpts
- Opening excerpt
- Pre-existing immunity to seasonal endemic coronaviruses could have profound consequences for antibody responses to SARS-CoV-2, induced from natural infection or vaccination.
- Closing excerpt
- Our results suggest that pre-existing immunity to endemic coronaviruses should be considered in evaluating antibody responses to SARS-CoV-2.
- Interpretation limits
- Verbatim excerpts from the authors’ abstract; the closing excerpt may describe a finding, limitation or recommendation. Design, population, effect size and uncertainty have not been independently extracted or appraised. Read the complete abstract and linked paper before interpreting the findings.
Read complete abstract (147 words)
Pre-existing immunity to seasonal endemic coronaviruses could have profound consequences for antibody responses to SARS-CoV-2, induced from natural infection or vaccination. A first step to establish whether pre-existing responses can impact SARS-CoV-2 infection is to understand the nature and extent of cross-reactivity in humans to coronaviruses. Here we compare serum antibody and memory B cell responses to coronavirus spike proteins from pre-pandemic and SARS-CoV-2 convalescent donors using binding and functional assays. We show weak evidence of pre-existing SARS-CoV-2 cross-reactive serum antibodies in pre-pandemic donors. However, we find evidence of pre-existing cross-reactive memory B cells that are activated during SARS-CoV-2 infection. Monoclonal antibodies show varying degrees of cross-reactivity with betacoronaviruses, including SARS-CoV-1 and endemic coronaviruses. We identify one cross-reactive neutralizing antibody specific to the S2 subunit of the S protein. Our results suggest that pre-existing immunity to endemic coronaviruses should be considered in evaluating antibody responses to SARS-CoV-2.
Abstract reproduced from the open-access article under the recorded cc by licence; © the authors. PMID 34011939 · DOI · PMC8134462
BCG-Induced Cross-Protection and Development of Trained Immunity: Implication for Vaccine Design [3]
Abstract reading summary — source excerpts
- Opening excerpt
- The Bacillus Calmette-Guérin (BCG) is a live attenuated tuberculosis vaccine that has the ability to induce non-specific cross-protection against pathogens that might be unrelated to the target disease.
- Closing excerpt
- In this review, we discuss BCG-induced cross-protection and acquisition of trained immunity and potential heterologous effects of recombinant BCG vaccines.
- Interpretation limits
- Verbatim excerpts from the authors’ abstract; the closing excerpt may describe a finding, limitation or recommendation. Design, population, effect size and uncertainty have not been independently extracted or appraised. Read the complete abstract and linked paper before interpreting the findings.
Read complete abstract (210 words)
The Bacillus Calmette-Guérin (BCG) is a live attenuated tuberculosis vaccine that has the ability to induce non-specific cross-protection against pathogens that might be unrelated to the target disease. Vaccination with BCG reduces mortality in newborns and induces an improved innate immune response against microorganisms other than Mycobacterium tuberculosis, such as Candida albicans and Staphylococcus aureus. Innate immune cells, including monocytes and natural killer (NK) cells, contribute to this non-specific immune protection in a way that is independent of memory T or B cells. This phenomenon associated with a memory-like response in innate immune cells is known as "trained immunity." Epigenetic reprogramming through histone modification in the regulatory elements of particular genes has been reported as one of the mechanisms associated with the induction of trained immunity in both, humans and mice. Indeed, it has been shown that BCG vaccination induces changes in the methylation pattern of histones associated with specific genes in circulating monocytes leading to a "trained" state. Importantly, these modifications can lead to the expression and/or repression of genes that are related to increased protection against secondary infections after vaccination, with improved pathogen recognition and faster inflammatory responses. In this review, we discuss BCG-induced cross-protection and acquisition of trained immunity and potential heterologous effects of recombinant BCG vaccines.
Abstract reproduced from the open-access article under the recorded cc by licence; © the authors. PMID 31849980 · DOI · PMC6896902
Cross-Protection in PRRSV: Mechanisms, Limitations, and Implications for Vaccine Design [4]
Abstract reading summary — source excerpts
- Opening excerpt
- Porcine reproductive and respiratory syndrome (PRRS) remains one of the most economically devastating diseases in global swine production.
- Closing excerpt
- Collectively, these findings highlight the multifactorial nature of PRRSV protection and the need for next-generation vaccines capable of inducing broader and more durable immunity.
- Interpretation limits
- Verbatim excerpts from the authors’ abstract; the closing excerpt may describe a finding, limitation or recommendation. Design, population, effect size and uncertainty have not been independently extracted or appraised. Read the complete abstract and linked paper before interpreting the findings.
Read complete abstract (192 words)
Porcine reproductive and respiratory syndrome (PRRS) remains one of the most economically devastating diseases in global swine production. The causative agent, PRRS virus (PRRSV), comprises two genetically distinct species-PRRSV-1 and PRRSV-2-that differ substantially in antigenic composition and immune recognition. Despite widespread use of modified live vaccines (MLVs), protection against heterologous and cross-species strains remains inconsistent and difficult to predict. This review synthesizes current knowledge of homologous, heterologous, and cross-species protection, with emphasis on humoral and cellular immune responses and the viral determinants that constrain breadth of immunity. Neutralizing antibodies can confer near-sterilizing homologous protection under controlled conditions; however, their delayed induction and narrow specificity limit efficacy against heterologous strains. T-cell-mediated responses are generally broader but remain highly strain- and context-dependent. Structural features of PRRSV envelope glycoproteins, including glycan shielding and immunodominant decoy epitopes, further restrict antibody-mediated cross-protection while providing targets for rational vaccine design. We also examine potential drawbacks of preexisting immunity, including antigenic mismatch and non-neutralizing antibody-dominated responses that may contribute to suboptimal outcomes following heterologous exposure. Collectively, these findings highlight the multifactorial nature of PRRSV protection and the need for next-generation vaccines capable of inducing broader and more durable immunity.
Abstract reproduced from the open-access article under the recorded cc by licence; © the authors. PMID 42075672 · DOI · PMC13119244
Key reviews and meta-analyses
The most cited review-type papers among PubMed’s best-match results for this heading, listed with bibliographic details only. Follow the links for the full text and access terms.
- Research article Non-specific effects of BCG vaccine on viral infections [5]
- Research article Cross-protective efficacy of two human papillomavirus vaccines: a systematic review and meta-analysis [6]
- Research article Conserved epitopes of influenza A virus inducing protective immunity and their prospects for universal vaccine development [7]
- Research article Virus-specific T cells as correlate of (cross-)protective immunity against influenza [8]
- Research article Mucosal IgA responses in influenza virus infections; thoughts for vaccine design [9]
- Research article Host Factors Impact Vaccine Efficacy: Implications for Seasonal and Universal Influenza Vaccine Programs [10]
References
- Gao Y, Cai C, Grifoni A, Müller TR, Niessl J, Olofsson A, et al.. Ancestral SARS-CoV-2-specific T cells cross-recognize the Omicron variant. . 2022. PMID 35042228 · DOI · PMC8938268
- Song G, He WT, Callaghan S, Anzanello F, Huang D, Ricketts J, et al.. Cross-reactive serum and memory B-cell responses to spike protein in SARS-CoV-2 and endemic coronavirus infection. . 2021. PMID 34011939 · DOI · PMC8134462
- Covián C, Fernández-Fierro A, Retamal-Díaz A, Díaz FE, Vasquez AE, Lay MK, et al.. BCG-Induced Cross-Protection and Development of Trained Immunity: Implication for Vaccine Design. . 2019. PMID 31849980 · DOI · PMC6896902
- Raev SA, Cai L, Muro N, Madera R, Wang L, Shi J. Cross-Protection in PRRSV: Mechanisms, Limitations, and Implications for Vaccine Design. . 2026. PMID 42075672 · DOI · PMC13119244
- Moorlag SJCFM, Arts RJW, van Crevel R, Netea MG. Non-specific effects of BCG vaccine on viral infections. Clin Microbiol Infect. 2019. PMID 31055165 · DOI
- Malagón T, Drolet M, Boily MC, Franco EL, Jit M, Brisson J, et al.. Cross-protective efficacy of two human papillomavirus vaccines: a systematic review and meta-analysis. Lancet Infect Dis. 2012. PMID 22920953 · DOI
- Staneková Z, Varečková E. Conserved epitopes of influenza A virus inducing protective immunity and their prospects for universal vaccine development. Virol J. 2010. PMID 21118546 · DOI · PMC3009981
- Altenburg AF, Rimmelzwaan GF, de Vries RD. Virus-specific T cells as correlate of (cross-)protective immunity against influenza. Vaccine. 2015. PMID 25498210 · DOI
- van Riet E, Ainai A, Suzuki T, Hasegawa H. Mucosal IgA responses in influenza virus infections; thoughts for vaccine design. Vaccine. 2012. PMID 22835738 · DOI
- Dhakal S, Klein SL. Host Factors Impact Vaccine Efficacy: Implications for Seasonal and Universal Influenza Vaccine Programs. J Virol. 2019. PMID 31391269 · DOI · PMC6803252
About this article
- Sources
- NLM Medical Subject Headings (definition, synonyms); NCBI PubMed (counts, identifiers); Europe PMC (open-access abstracts and citation counts).
- Verification
- Every reference was resolved against PubMed on 2026-10-31; title, year and DOI matched.
- Review state
- automated source verified; no human expert review has been recorded yet.
- Version
- 1, last updated 2026-10-29.
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Evidence strength
EvidenceHelix's computed view of the study designs behind this topic: how common meta-analyses, randomized trials and reviews are, how recent the literature is, and a plain-language reading. It describes the design mix of the literature. It is not a quality appraisal of any study, and it is not medical advice.